مقالات پذیرفته شده در هشتمین کنگره بین المللی زیست پزشکی
The effect of Inflammation and opportunistic microbiomes on colon cancer
The effect of Inflammation and opportunistic microbiomes on colon cancer
Sahar Hemati,1Mahtab Maleki,2,*
1. Department of biology, Islamshahr Branch, Islamic Azad University, Tehran, Iran 2. Department of Biology, Science and Research Branch , Islamic Azad University , Tehran , Iran
Introduction: The second most common cause of death worldwide Is cancer, and it is widely
known that bacteria play a role in a number of diseases, including cancer. It is well
recognized that the gut microbiota is crucial to the physiology and overall health of
the host. Opportunistic bacteria in the gut microbiota can cause infection, while
helpful bacteria produce essential nutrients like vitamin B and K. Dysbiosis, caused
by imbalances between these groups, is linked to gastrointestinal conditions like
colorectal cancer (CRC) and inflammatory bowel disease (IBD). The gut microbiota
may also play a role in the pathophysiology of inflammatory bowel disease (IBD), as
host reactions to gut bacteria are correlated with specific genes. Inflammatory bowel
disease (IBD) is a condition characterized by a decrease in microbial diversity,
resulting in an increase in pathogenic bacteria and a decrease in helpful bacteria.
The microbiota of individuals with inflammatory bowel disease (IBD) is marked by a
reduction in bacterial species, a decrease in Firmicutes and Bacteroidetes, and an
increase in Proteobacteria. The compromised mucus layer allows luminal bacteria to
enter submucosal layers, triggering proliferative and inflammatory processes.
Dysbiosis and a surge in bacteria, such as Bacteroides fragilis, which disrupts the
intestinal mucosal barrier, allow more bacteria to pass from the lumen to the interior
of the tissue. Due to the chronic tissue inflammation caused by this condition, pro-
carcinogenic and inflammatory mediators are released, increasing the risk of
colorectal cancer (CRC). The innovative approach to treatment of colorectal cancer
involves utilizing probiotics and prebiotics to manipulate the gut flora, improving gut
barrier function, immune modulation, and colonization resistance. Probiotics and
prebiotics can enhance anticancer therapies by altering gut microbiota metabolism.
To effectively treat colon cancer, new bacterial species should be developed as
probiotic supplements based on each patient's unique genetic background.
Methods: We used keywords like intestinal microbiome, colorectal cancer, immune system and
microbiota interaction, and probiotics to search the databases of Google Scholar,
PubMed, Nature, and Scopus in order to write this review article. 57 English-
language articles were selected among the many articles obtained according to the
content, quality, and credibility of the magazine. The selected articles were divided
into three groups.The microbiome and its affecting variables were Introduced in the
first set of articles. The second set of articles addressed therapeutic approaches as
well as the connection between human diseases and the microbiome. The effects of
probiotics were discussed in the third set of publications.
Results: The vast population of bacteria that make up the gut microbiota is frequently referred
to as "a forgotten argan" in relation to human health and illnesses. There is growing
evidence linking colorectal cancer (CRC) to dysbiosis of the gut microbiota. It is
getting more and more obvious what functions gut microbes played in both starting
and facilitating the colorectal cancer (CRC) process. A variety of hypothesis models
have been put forth to illustrate the intricate connection between gut microbiota and
colorectal cancer. Colorectal cancer (CRC) prevention strategies utilize various
tactics, including prebiotics and probiotics, to manipulate gut microbiota, aiming to
reverse microbial dysbiosis and improve overall health.
Conclusion: Colorectal cancer (CRC) onset and gut microbiota dysbiosis are strongly correlated,
as we have highlighted. Colorectal cancer (CRC) is made more likely by certain
bacteria that can induce intestinal inflammation. Contrarily, several bacteria have the
ability to create substances that improve the Intestinal mucosal barrier's ability to
prevent the development of colorectal cancer (CRC). One of the most promising new
approaches in medicine to enhance people's health is microbiome regulation. Thus,
further studies and clinical trials pertaining to the gut microbiome It is necessary to
assess the effectiveness of systemic treatments for colorectal cancer (CRC), reduce
side effects, and raise survival rates in both oncology and colorectal cancer (CRC)
patient care.
Keywords: Gut microbiota, dysbiosis, colorectal cancer (CRC), inflammatory bowel disease
(IBD), probiotic, pre